Summary statistics are available for download from the GWAS Catalog.
A Genome-Wide Association Study of Diabetic Kidney Disease in Subjects With Type 2 Diabetes.
van Zuydam NR, et al.
Diabetes. 2018 Jul;67(7):1414-1427. doi: 10.2337/db17-0914
Summary statistics are available for download from the GWAS Catalog.
A Genome-Wide Association Study of Diabetic Kidney Disease in Subjects With Type 2 Diabetes.
van Zuydam NR, et al.
Diabetes. 2018 Jul;67(7):1414-1427. doi: 10.2337/db17-0914
Summary statistics are available for download from the GWAS Catalog.
A Genome-Wide Association Study of Diabetic Kidney Disease in Subjects With Type 2 Diabetes.
van Zuydam NR, et al.
Diabetes. 2018 Jul;67(7):1414-1427. doi: 10.2337/db17-0914
Exome sequencing identifies rare LDLR and APOA5 alleles conferring risk for myocardial infarction.
Do R, et al.
Nature. 2015 Feb 5;518(7537):102-6. doi: 10.1038/nature13917
Exome sequencing identifies rare LDLR and APOA5 alleles conferring risk for myocardial infarction.
Do R, et al.
Nature. 2015 Feb 5;518(7537):102-6. doi: 10.1038/nature13917
Exome sequencing identifies rare LDLR and APOA5 alleles conferring risk for myocardial infarction.
Do R, et al.
Nature. 2015 Feb 5;518(7537):102-6. doi: 10.1038/nature13917
Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity.
Turcot V, et al.
Nat Genet. 2018 Jan;50(1):26-41. doi: 10.1038/s41588-017-0011-x
Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity.
Turcot V, et al.
Nat Genet. 2018 Jan;50(1):26-41. doi: 10.1038/s41588-017-0011-x
Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity.
Turcot V, et al.
Nat Genet. 2018 Jan;50(1):26-41. doi: 10.1038/s41588-017-0011-x
Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity.
Turcot V, et al.
Nat Genet. 2018 Jan;50(1):26-41. doi: 10.1038/s41588-017-0011-x